Sports Concussions, Dementia, and APOE Genotyping: What Can Scientists Tell the Public? What’s Up for Research?
the best large population data out there. See Guskiewicz et al., 2005 and Guskiewicz et al., 2007.
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Mice on Trial? Issues in the Design of Drug Studies
neurological disease is the discovery by Hauser et al., recently published in the New England Journal of
Targeting Tumor Necrosis Factor—A Therapeutic Strategy for AD
inhibitor, which blocks its translocation: dehydroxymethylepoxyquinomicin (Kubota et al., 2007). Lovastatin
Non-coding RNAs in Neurodegeneration
using different techniques, this might be a reasonable resource (Wu et al., 2007). Kenneth Kosik Once
The AD8: A Brief Screening Tool for Very Mild Dementia
(Galvin et al., 2005). Brief cognitive tests such as the Mini Mental State Examination (MMSE) may help
Susceptibility Testing and Risk Assessment in Alzheimer Disease
dementia patient. The Neurologist, 1997. 3: p. 13-30. 2. Strittmatter, W.J., et al., Apolipoprotein E:
Finding NIH Support for Drug Discovery and Development for Neurodegenerative Diseases
Searching for the wherewithal to push on with your work on a promising new Alzheimer therapy? The NIH offers funding for research support at every step of the process, from early drug discovery right through to phase 3 clinical trials, if you know where t
Presenilin Loss of Function—Plan B for AD?
dying in droves in an environment of gliosis (Saura et al., 2004). These mice have no amyloid, but they
ADCS Prevention Instrument Project
appeared in the October-December 2006 supplement. Steve Ferris et al. 0 WEBINAR Monday, March 12, 2007-
The Functional Roles of APP Cytoplasmic Domain—Conflicts and Consensus
Fe65 knockout (KO) mice (see Guenette et al., 2006) and Ulrike Muller showed with her APP triple KO
Webinar: Axonopathy in Mouse Models of AD
transgenic mouse models (Casas et al., 2004). Some neurodegenerative diseases including AD and amyotrophic
Messing with the Membrane—An Alternative Interpretation of the Amyloid-β Hypothesis
genetic burden has as yet been described (Oddo et al., 2003). The triple transgenic mouse strain confirmed
Cell Cycle Hypothesis Pedaling into Mainstream Acceptance? Results in Fly, Mouse Models Warrant a Second Look
tau-positive. Was the reverse true? I ask because if you look at our AD study (Busser et al., 1998), we found
Gain or Loss of Function—Time to Shake up Assumptions on γ-Secretase in Alzheimer Disease?
Aβ40 levels whereas only some drove up Aβ42. Kumar-Singh et al. also found that loss of Ab40 correlated
Not Dead Yet: Estrogen Deserves Another Chance
itself and has brain-specific aromatase (see Sasano et al., 1998). Dominique Toran-Allerand Regarding
Have a topic idea for a webinar? We would love to hear it. Send an email to webinars [at] alzforum [dot] org.
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